Whether emf blocking live in an apartment or house, or you just want to keep your home free of EMFs There are a variety of ways you can reduce exposure. One of the most effective is to reduce the use of electronic devices. You could also consider EMF blocker paint to block EMF radiation from entering your house. emf blocker to shield your home from EMF radiation would be to put up a shielding canopy for RF. It is a type made of net that has EMF shielding. It's used to stop EMFs from entering a space. Another option is to get your home equipped with a conductive enclosure. They are referred to as Faraday cages.

Numerous studies have demonstrated studies have shown that nonionizing EMF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. This could explain the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM EMF's effects on cancer patients is not yet clear.

The effects from AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were divided into 3 groups. One group did not have exposure RF EMF. Second group members were exposed to RF EMF at the same frequency to that used in humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumors was assessed against the effect of RCF. emf blocking indicated that tumours treated with HCCMF had significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage in the tumour.

The mechanism behind tumour-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is mediated through CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have more broader implications in the treatment and diagnosis of different cancers.

The tumors in the controls were never exposed RF EMF, and were fed a normal mouse diet. The tumours in those in the HCCMF group were injected with Huh7 cells after they were five-seven weeks old. The tumors were removed in cases of excessive burden.


The tumors of the three groups also showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, the tumors treated with RCF showed no shrinkage. The difference was significant. The tumors treated with RCF had necrosis that is common when tumors are that are exposed to RCF. The possibility is that this necrosis was caused by the lack of oxygen in larger tumors.

In summary, the results indicate an AM-RF EMF exhibits anticancer activity in vitro and in live. Numerous studies have demonstrated that AM RF EMF produces measurable reduction in tumours in HCC patients. The possibility is that the AM EMF produces these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained influence on the development of HCC tumours in vivo.